267 research outputs found
A note on q-Euler numbers and polynomials
The purpose of this paper is to construct q-Euler numbers and polynomials by
using p-adic q-integral equations on Zp. Finally, we will give some interesting
formulae related to these q-Euler numbers and polynomials.Comment: 6 page
Synthesis of tacrine analogues derived from n-aryl-5-amino-4-cyanopyrazoles
Synthesis of eleven tacrine analogues derived from N-aryl-5-amino-4-cyanopyrazoles, by a Friedländer type reaction, is described. Their structures were confirmed by 1H and 13C NMR spectroscopy, elemental analysis and/or mass spectrometry.Fundação para a Ciência e Tecnologia and FEDER (POCTI-SFA-3-686
A Selberg integral for the Lie algebra A_n
A new q-binomial theorem for Macdonald polynomials is employed to prove an
A_n analogue of the celebrated Selberg integral. This confirms the g=A_n case
of a conjecture by Mukhin and Varchenko concerning the existence of a Selberg
integral for every simple Lie algebra g.Comment: 32 page
Block-Anti-Circulant Unbalanced Oil and Vinegar
We introduce a new technique for compressing the public keys of the UOV signature scheme that makes use of block-anti-circulant matrices. These matrices admit a compact representation as for every block, the remaining elements can be inferred from the first row. This space saving translates to the public key, which as a result of this technique can be shrunk by a small integer factor. We propose parameters sets that take into account several important attacks
Simple deterministic dynamical systems with fractal diffusion coefficients
We analyze a simple model of deterministic diffusion. The model consists of a
one-dimensional periodic array of scatterers in which point particles move from
cell to cell as defined by a piecewise linear map. The microscopic chaotic
scattering process of the map can be changed by a control parameter. This
induces a parameter dependence for the macroscopic diffusion coefficient. We
calculate the diffusion coefficent and the largest eigenmodes of the system by
using Markov partitions and by solving the eigenvalue problems of respective
topological transition matrices. For different boundary conditions we find that
the largest eigenmodes of the map match to the ones of the simple
phenomenological diffusion equation. Our main result is that the difffusion
coefficient exhibits a fractal structure by varying the system parameter. To
understand the origin of this fractal structure, we give qualitative and
quantitative arguments. These arguments relate the sequence of oscillations in
the strength of the parameter-dependent diffusion coefficient to the
microscopic coupling of the single scatterers which changes by varying the
control parameter.Comment: 28 pages (revtex), 12 figures (postscript), submitted to Phys. Rev.
Restricted Expression of Epstein-Barr Virus Latent Genes in Murine B Cells Derived from Embryonic Stem Cells
Background: Several human malignancies are associated with Epstein-Barr virus (EBV) and more than 95 % of the adult human population carries this virus lifelong. EBV efficiently infects human B cells and persists in this cellular compartment latently. EBV-infected B cells become activated and growth transformed, express a characteristic set of viral latent genes, and acquire the status of proliferating lymphoblastoid cell lines in vitro. Because EBV infects only primate cells, it has not been possible to establish a model of infection in immunocompetent rodents. Such a model would be most desirable in order to study EBV’s pathogenesis and latency in a suitable and amenable host. Methodology/Principal Findings: We stably introduced recombinant EBV genomes into mouse embryonic stem cells and induced their differentiation to B cells in vitro to develop the desired model. In vitro differentiated murine B cells maintained the EBV genomes but expression of viral genes was restricted to the latent membrane proteins (LMPs). In contrast to human B cells, EBV’s nuclear antigens (EBNAs) were not expressed detectably and growth transformed murine B cells did not arise in vitro. Aberrant splicing and premature termination of EBNA mRNAs most likely prevented the expression of EBNA genes required for B-cell transformation. Conclusions/Significance: Our findings indicate that fundamental differences in gene regulation between mouse and ma
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